Bibliografia
[1] Ngudi, Delphin Diasolua, et al. "Research on motor neuron diseases konzo and neurolathyrism: trends from 1990 to 2010." PLoS neglected tropical diseases6.7 (2012): e1759.
[2] Singh, Surya S., and S. L. N. Rao. "Lessons from neurolathyrism: A disease of the past & the future of Lathyrus sativus (Khesari dal)." The Indian journal of medical research 138.1 (2013): 32.
[3] Kusama‐Eguchi, Kuniko, et al. "Hind‐limb paraparesis in a rat model for neurolathyrism associated with apoptosis and an impaired vascular endothelial growth factor system in the spinal cord." Journal of Comparative Neurology 518.6 (2010): 928-942.
[4] Kuniko Kusama-Eguchi, et al. "New insights into the mechanism of neurolathyrism: L-β-ODAP triggers [Ca2+]i accumulation and cell death in primary motor neurons through transient receptor potential channels and metabotropic glutamate receptors." Food and Chemical Toxicology 67 (2014): 113-122.
[5] Barceloux, D.G. "Grass pea and neurolathyrism (Lathyrus sativus L.)" Disease-a-month 55.6 (2009): 365-372.
[6] Khandare, Arjun L., M. Ankulu, and N. Aparna. "Role of glutamate and nitric oxide in onset of motor neuron degeneration in neurolathyrism." Neurotoxicology 34 (2013): 269-274.
[7] Khandare, Arjun L., M. Ankulu, and N. Aparna. "Role of glutamate and nitric oxide in onset of motor neuron degeneration in neurolathyrism."Neurotoxicology 34 (2013): 269-274.
[8] Chang, Louis W., and William Slikker Jr, eds. Neurotoxicology: approaches and methods. academic press, 1995.
β-ODAP
Ácido β-N-oxalil-L-α,β-diaminopropriónico
